当社グループは 3,000 以上の世界的なカンファレンスシリーズ 米国、ヨーロッパ、世界中で毎年イベントが開催されます。 1,000 のより科学的な学会からの支援を受けたアジア および 700 以上の オープン アクセスを発行ジャーナルには 50,000 人以上の著名人が掲載されており、科学者が編集委員として名高い
。オープンアクセスジャーナルはより多くの読者と引用を獲得
700 ジャーナル と 15,000,000 人の読者 各ジャーナルは 25,000 人以上の読者を獲得
Ulfuara Shefa
Aerophilic injury is powerfully concerned within the pathologic process of neurodegenerative maladys as well as Alzheimer’s disease, amyotrophic lateral pathology, Huntington’s malady, Parkinson’s malady and stroke (brain ischemia/reperfusion injury). The supply of transgenic and toxin-inducible models of those conditions has expedited the diagnosing analysis of supposed inhibitor agents starting from prototypical natural antioxidants like fat-soluble vitamin (α-tocopherol) to subtle artificial radical traps and chemical action oxidants. Literature review shows that inhibitor therapies have enjoyed general success in diagnosing studies across disparate animal models, however very little profit in human intervention studies or clinical trials. Recent high-profile failures of fat-soluble vitamin trials in Parkinson’s malady, and nitrone therapies in stroke, have diminished enthusiasm to pursue inhibitor neuro protectants within the clinic. The change of location disappointment of antioxidants probably arises from a mix of things as well as failure to grasp the drug candidate’s mechanism of action in relationship to human malady, and failure to conduct diagnosing studies mistreatment concentration and time parameters relevant to the clinical setting. This review discusses the explanation for mistreatment antioxidants within the prevention or mitigation of human neuro diseases, with a vital discussion concerning ways that within which future diagnosing studies could also be adjusted to supply a lot of prophetic worth in choosing agents for translation into human trials.