当社グループは 3,000 以上の世界的なカンファレンスシリーズ 米国、ヨーロッパ、世界中で毎年イベントが開催されます。 1,000 のより科学的な学会からの支援を受けたアジア および 700 以上の オープン アクセスを発行ジャーナルには 50,000 人以上の著名人が掲載されており、科学者が編集委員として名高い
。オープンアクセスジャーナルはより多くの読者と引用を獲得
700 ジャーナル と 15,000,000 人の読者 各ジャーナルは 25,000 人以上の読者を獲得
Surena Vahabi*, Shahab Kavousinejad , Ghazal Ranjbar , Azadeh Ghasemi and Fatemeh Alirezaei
Background and aim: Alzheimer is a cognitive disorder which has irreversible conditions, and causes difficulties in judgment and decision. The main mechanism of the disease would be through the inflammation in the brain which probably has a complex interaction with the environment, cardiology and genetics, so that the present information reinforces the idea that the periodontal pathogens are the potential factors for inflammation of neurons and the occurrence of Alzheimer. Periodontitis is a common source of chronic systemic infection illness which in this junctional epithelium and periodontal pockets disease can be a conduit entry for bacteria into the bloodstream. Periodontopathic bacteria and systemic inflammation caused by them act as a bridge between these diseases and Alzheimer.
Materials and methods: The research was conducted by review. A literature review was performed by referring to the scientific papers on the internet from the PubMed and Google Scholar databases, between 1985 and 2016.
Results: Periodontal disease influences the Alzheimer’s disease through various mechanisms. It can be said that periodontal disease by pathogenic bacteria involved in periodontal inflammatory molecules that cause brain inflammation associated with Alzheimer. P. gingivalis with penetration to the CNS causes dementia. Vitamin B12 deficiency is also associated with cognitive disorders.